![]() ![]() ![]() Together with central command and arterial/cardiopulmonary baroreceptors, groups III and IV skeletal muscle nerve afferents play an important role in mediating hemodynamic responses to exercise. On the other hand, the metabolite accumulation due to BFR also increases afferent signaling of group IV afferent muscle nerves ( Nobrega et al., 2014). These effects have been associated to increased metabolite accumulation in the active skeletal muscle ( Suga et al., 2012 Pearson and Hussain, 2015). Resistance training with low to moderate loads performed under blood flow restriction (BFR) has been shown to elicit muscle hypertrophy and strength gains ( Kaijser et al., 1990 Abe et al., 2006 Manini and Clark, 2009 Takada et al., 2012). However, MAP reduction during handgrip seemed not to be provoked by lowered metaboreflex activity. Additionally, none of the observed hemodynamic outcomes, including systemic vascular resistance (SVR), showed significant difference between T0 and T1 during the metaboreflex activation ( P > 0.05).Ĭonclusion: BFRT reduced blood pressure during handgrip exercise, thereby suggesting a potential hypotensive effect of this modality of training. ![]() However, no significant time effect was detected for MAP during the metaboreflex activation ( P > 0.05). Results: BFRT reduced MAP during handgrip exercise (T1: 96.3 ± 8.3 mmHg vs. Hemodynamic measurements were assessed by means of impedance cardiography. On the first week, the occlusion was set at 75% of resting systolic blood pressure (always obtained after 3 min of resting) and increased 25% every week, until reaching 150% of resting systolic pressure at week four. BFRT consisted of 3-min rhythmic handgrip exercise applied 3 days/week (30 contractions per minute at 30% of maximum voluntary contraction) in the dominant arm. Hemodynamic responses during muscle metaboreflex were assessed by means of postexercise muscle ischemia (PEMI) at baseline (T0) and after 1 month (T1) of dynamic BFRT. Methods: 17 young healthy males aged 18–25 yrs enrolled in this study. Then, we hypothesized that a period of BFR training would result in blunted hemodynamic activation during muscle metaboreflex. However, BFR training also causes metabolite accumulation that may desensitize type III and IV nerve endings, which trigger muscle metaboreflex. ![]() Objective: Blood flow restriction training (BFRT) has been proposed to induce muscle hypertrophy, but its safety remains controversial as it may increase mean arterial pressure (MAP) due to muscle metaboreflex activation.
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